Crop Circle Architecture May Provide Anti Viral Information Against International Pandemic

Are we being watched, monitored, coached by our space brothers?

 

On August 19, 2020, Frank Joseph, author, joined Jeff Rense, talk show host, to discuss a recent 200 foot wide crop circle found in England that they believe emulates the architecture of the coronavirus.  The crop circle shows one enlarged portion of the virus, that Rense and Joseph, believe might possibly depict an obvious starting point to consider for annihilation of the virus.

 

Link to YouTube video “Jeff & Frank Joseph – Did ET Crop Circles Just Cure Coronavirus?” – https://www.youtube.com/watch?v=QaU7_hyFXrw&feature=youtu.be

 

 

About twenty miles away, another crop circle depicting, according to Joseph, a compound used to ward off pathogens; such as Corona: 2,4-Dimethyl-3-isopropylpentane.

 

 

Our immune system may be able to provide support against an entire series of corona based pathogens.   PubChem lists Dimethyl-3-isopropylpentane as an anti Viral.

2,4-Dimethyl-3-isopropylpentane information below from PubChem.

 

The BioAcoustically based frequency decoded for 2,4-Dimethyl-3-isopropylpentane turned out to be an identical mathematical equivalent to Cytotoxic T-Lymphocyte Antigen 4 (CTLA-4).

 

BioAcoustically Speaking, frequencies can pitch-hit for each other.

 

It is incredible that:

-A crop circle could depict a pathogen threatening the earth

-A potential antiviral hydrocarbon crop circle nearby could indicate a potential cure for the pathogen threatening the earth

-That the hydrocarbon indicated by the crop circle could have identical mathematical properties to a member of a known immunoglobulin superfamily already known by scientists to be involved in regulating the body’s immune responses.

 

FROM WIKIPEDIA Cytotoxic T-Lymphocyte Antigen 4 (CTLA-4 CTLA4) is a member of the immunoglobulin superfamily that is expressed by activated T cells and transmits an inhibitory signal to T cells. CTLA4 is homologous to the T-cell co-stimulatory protein, CD28, and both molecules bind to CD80 and CD86, also called B7-1 and B7-2 respectively, on antigen-presenting cells. CTLA-4 binds CD80 and CD86 with greater affinity and avidity than CD28 thus enabling it to outcompete CD28 for its ligands. CTLA4 transmits an inhibitory signal to T cells, whereas CD28 transmits a stimulatory signal. CTLA4 is also found in regulatory T cells (Tregs) and contributes to their inhibitory function. T cell activation through the T cell receptor and CD28 leads to increased expression of CTLA-4.

 

 

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Frequencies Suggested:

Ac   Bc

13.34/22.23

15.56/20.01

17.51/18.06

 

Use this listing of beginning frequencies in a binaural manner using an online frequency generator: www.OnlineToneGenerator.com

 

What people can do:

1. Download Le Ciel created by James Marshall, actor/composer. James is working on a CV specific piece of music that we want to make available ASAP. He hopes to finish the piece by the end of this week. Access the music here: https://soundhealthoptions.com/product/le-ciel-8-minute-version/

   You can also download Jill Mattson’s Corona Defense music here: coronavirusdefense.org
   Jill created meditation music with the Corona Conflicts frequencies in the background – there are many versions, download your favorite.

 

2. A Corona Conflicts BioAcoustic evaluation is available online 24/7 – SoundHealthPortal.com offers individual vocal profiles dealing with the information we have amassed so far. BioAcoustically monitor your nutritional and immune system requirements under the SERVICES tab.

   An additional BioAcoustic BioMarker bundle has been added that includes information about Corona Conflicts, Immune System, allergy responses, individual energy and metabolic systems, radiation exposures and inflammatory status.  Link to demonstration: https://vimeo.com/396458125

 

3. Use this listing of beginning frequencies in a binaural manner using an online frequency generator: www.OnlineToneGenerator.com.

 a. Use at your own risk. Use biofeedback of oxygen and heart monitoring to check for positive/negative reactions. Link: https://soundhealthoptions.com/wpcontent/uploads/2019/11/LeCiel-NegReactions.pdf

 

b. If you can’t hear the tones of your device. Simply double the frequencies until the tone are perceptible.

 

-For best results only each set of tones separately using the binaural setting.

-Each tone should be played no more than one minute per tone

-Play each tone through twice, every 2-3 hours or as comfortable

-Tones should be low in volume, barely audible

-Lower frequency should always be on the A Channel (Ac)

-Safely use under the guidance of a trained supervisor

 

Sharry Edwards, MEd., Director

Institute of BioAcoustic Biology & Sound Health

 

August 26, 2020 – © Sharry Edwards, MEd

 

Additional Information  – 2,4-Dimethyl-3-isopropylpentane

 

Patent info:  https://europepmc.org/article/PAT/US2009176730  – N4-ACYLCYTOSINE NUCLEOSIDES FOR TREATMENT OF VIRAL INFECTIONS

The present invention is directed to a method and composition of treating or preventing viral infections, in particular, human immunodeficiency virus (HIV) and hepatitis B virus (HBV) infections, in human patients or other animal hosts, comprising the administration of N.sup.4-acyl-2′,3′-dideoxy-5-fluorocytidine or N.sup.4-acyl-2′,3′-didehydro-2′,3′-dideoxy-5-fluorocytidine, and pharmaceutically acceptable salts, prodrugs, and other derivatives thereof.

CTLA-A-4 – Function

WIKIPEDIA:  The mechanism by which CTLA-4 acts in T cells remains somewhat controversial. Biochemical evidence suggested that CTLA-4 recruits a phosphatase to the T cell receptor (TCR), thus attenuating the signal. This work remains unconfirmed in the literature since its first publication. More recent work has suggested that CTLA-4 may function in vivo by capturing and removing B7-1 and B7-2 from the membranes of antigen-presenting cells, thus making these unavailable for triggering of CD28.

In addition to that, it has been found that dendritic cell (DC) – Treg interaction causes sequestration of Fascin-1, an actin-bundling protein essential for immunological synapse formation and skews Fascin-1–dependent actin polarization in antigen presenting DCs toward the Treg cell adhesion zone. Although it is reversible upon T regulatory cell disengagement, this sequestration of essential cytoskeletal components causes a lethargic state of DCs, leading to reduced T cell priming. This suggests Treg-mediated immune suppression is a multi-step process. In addition to CTLA-4 CD80/CD86 interaction, fascin-dependent polarization of the cytoskeleton towards DC-Treg immune synapse may play a pivotal role.^[17]

CTLA-4 may also function via modulation of cell motility and/or signaling through PI3 kinase^[18] Early multiphoton microscopy studies observing T-cell motility in intact lymph nodes appeared to give evidence for the so-called ‘reverse-stop signaling model’.^[19] In this model CTLA-4 reverses the TCR-induced ‘stop signal’ needed for firm contact between T cells and antigen-presenting cells (APCs).^[20] However, those studies compared CTLA-4 positive cells, which are predominantly regulatory cells and are at least partially activated, with CTLA-4 negative naive T cells. The disparity of these cells in multiple regards may explain some of these results. Other groups who have analyzed the effect of antibodies to CTLA-4 in vivo have concluded little or no effect upon motility in the context of anergic T-cells.^[21] Antibodies to CTLA-4 may exert additional effects when used in vivo, by binding and thereby depleting regulatory T cells.^[22]

 

Clinical significance[edit]

Variants in this gene have been associated with Type 1 diabetes, Graves’ disease, Hashimoto’s thyroiditis, celiac disease, systemic lupus erythematosus, thyroid-associated orbitopathy, primary biliary cirrhosis and other autoimmune diseases.

Polymorphisms of the CTLA-4 gene are associated with autoimmune diseases such as rheumatoid arthritis^[23], autoimmune thyroid disease and multiple sclerosis, though this association is often weak. In systemic lupus erythematosus (SLE), the splice variant sCTLA-4 is found to be aberrantly produced and found in the serum of patients with active SLE.